Agnes Lukasik 

Research Assistant / Mental Health First Aider / Wellbeing Advocate / Disabled Staff Network committee member / EDIW committee member   

           

Email: al473@medschl.cam.ac.uk

The main focus of my work is to identify molecular mechanisms involved in controlling energy expenditure, fat deposition, and the partitioning of energy towards either oxidation or storage.

I first developed my interest in science through studying for a Master of Science in Biotechnology at the West Pomeranian University of Technology in Szczecin, Poland. After that, I joined the IMS-MRL and have been working here for nearly a decade, based in the TVPLab. 

Over the last few years, my role has evolved to include many aspects focused on mental health and wellbeing. As a wellbeing advocate and Mental Health First Aider, I am point of contact and support for staff and students. I am always there to support people as I am warm, non-judgmental and discrete.

In my current role, I come across people of different ages and nationalities who all hold different positions within the department. I am sensitive to how difficult it is for people to move to new environment. Many new staff members or students who join the IMS-MRL are not only coming to a new city but to a new country. I always try help to make their move as pleasant as possible. 

I am responsible for  designed, development and maintenance of  TVPLab website and social media.

 

My Awards

-Level 3 Certificate in Counselling, AIM,

-Certificate in Awareness of Mental Health Problems, 

-Adult Mental Health First Aider,

Publications

Suppression of insulin-induced gene 1 (INSIG1) function promotes hepatic lipid remodelling and restrains NASH progression. Azzu V, Vacca M, Kamzolas I, Hall Z, Leslie J, Carobbio S, Virtue S, Davies SE, Lukasik A, Dale M, Bohlooly-Y M, Acharjee A, Lindén D, Bidault G, Petsalaki E, Griffin JL, Oakley F, Allison MED, Vidal-Puig A. Mol Metab. 2021 Jun; 48:101210.

 

Defective peroxisomal proliferators activated receptor gamma activity due to dominant-negative mutation synergizes with hypertension to accelerate cardiac fibrosis in mice. Kis A, Murdoch C, Zhang M, Siva A, Rodriguez-Cuenca S, Carobbio S, Lukasik A, Blount M, O'Rahilly S, Gray SL, Shah AM, Vidal-Puig A. Eur J Heart Fail. 2009 Jun;11(6): 533-41

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